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paw grip strength  (Columbus Instruments)


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    Structured Review

    Columbus Instruments paw grip strength
    ( A ) Schematic representation of experimental design. Twelve-week-old male C57BL/6 were injected with KPC cells or PBS into the pancreas. On day 21, hindlimb muscles of the mice were isolated and processed for morphometric, biochemical, and snRNA-Seq analyses. ( B and C ) Changes in absolute body (BW) weight ( B ), and tumor-free BW ( C ) in control and KPC tumor–bearing mice. ( D <t>)</t> <t>Four-paw</t> grip strength normalized by tumor-free BW of control and KPC tumor–bearing mice. ( E ) Representative anti-laminin– and DAPI-stained TA muscle transverse sections. Scale bar: 50 μm. ( F ) Quantification of average myofiber cross-sectional area (CSA) in TA muscle of control and KPC tumor–bearing mice. n = 5 per group. All data are presented as mean ± SEM. * P ≤ 0.05, significantly different from control mice injected with PBS alone analyzed by unpaired Student’s t test. ( G ) UMAP plot representing manually annotated clusters for cell type identity. ( H ) Expression of representative marker genes define major cell types in dot plot. ( I ) Split-UMAP illustrating transcriptomic clustering of muscle-derived nuclei of control and KPC tumor–bearing mice. ( J ) Proportion of different nuclei population in GA muscle of control and KPC tumor–bearing mice.
    Paw Grip Strength, supplied by Columbus Instruments, used in various techniques. Bioz Stars score: 96/100, based on 1479 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/paw grip strength/product/Columbus Instruments
    Average 96 stars, based on 1479 article reviews
    paw grip strength - by Bioz Stars, 2026-06
    96/100 stars

    Images

    1) Product Images from "Fiber-type vulnerability and proteostasis reprogramming in skeletal muscle during pancreatic cancer cachexia"

    Article Title: Fiber-type vulnerability and proteostasis reprogramming in skeletal muscle during pancreatic cancer cachexia

    Journal: JCI Insight

    doi: 10.1172/jci.insight.200396

    ( A ) Schematic representation of experimental design. Twelve-week-old male C57BL/6 were injected with KPC cells or PBS into the pancreas. On day 21, hindlimb muscles of the mice were isolated and processed for morphometric, biochemical, and snRNA-Seq analyses. ( B and C ) Changes in absolute body (BW) weight ( B ), and tumor-free BW ( C ) in control and KPC tumor–bearing mice. ( D ) Four-paw grip strength normalized by tumor-free BW of control and KPC tumor–bearing mice. ( E ) Representative anti-laminin– and DAPI-stained TA muscle transverse sections. Scale bar: 50 μm. ( F ) Quantification of average myofiber cross-sectional area (CSA) in TA muscle of control and KPC tumor–bearing mice. n = 5 per group. All data are presented as mean ± SEM. * P ≤ 0.05, significantly different from control mice injected with PBS alone analyzed by unpaired Student’s t test. ( G ) UMAP plot representing manually annotated clusters for cell type identity. ( H ) Expression of representative marker genes define major cell types in dot plot. ( I ) Split-UMAP illustrating transcriptomic clustering of muscle-derived nuclei of control and KPC tumor–bearing mice. ( J ) Proportion of different nuclei population in GA muscle of control and KPC tumor–bearing mice.
    Figure Legend Snippet: ( A ) Schematic representation of experimental design. Twelve-week-old male C57BL/6 were injected with KPC cells or PBS into the pancreas. On day 21, hindlimb muscles of the mice were isolated and processed for morphometric, biochemical, and snRNA-Seq analyses. ( B and C ) Changes in absolute body (BW) weight ( B ), and tumor-free BW ( C ) in control and KPC tumor–bearing mice. ( D ) Four-paw grip strength normalized by tumor-free BW of control and KPC tumor–bearing mice. ( E ) Representative anti-laminin– and DAPI-stained TA muscle transverse sections. Scale bar: 50 μm. ( F ) Quantification of average myofiber cross-sectional area (CSA) in TA muscle of control and KPC tumor–bearing mice. n = 5 per group. All data are presented as mean ± SEM. * P ≤ 0.05, significantly different from control mice injected with PBS alone analyzed by unpaired Student’s t test. ( G ) UMAP plot representing manually annotated clusters for cell type identity. ( H ) Expression of representative marker genes define major cell types in dot plot. ( I ) Split-UMAP illustrating transcriptomic clustering of muscle-derived nuclei of control and KPC tumor–bearing mice. ( J ) Proportion of different nuclei population in GA muscle of control and KPC tumor–bearing mice.

    Techniques Used: Injection, Muscles, Isolation, Control, Staining, Expressing, Marker, Derivative Assay



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    ( A ) Schematic representation of experimental design. Twelve-week-old male C57BL/6 were injected with KPC cells or PBS into the pancreas. On day 21, hindlimb muscles of the mice were isolated and processed for morphometric, biochemical, and snRNA-Seq analyses. ( B and C ) Changes in absolute body (BW) weight ( B ), and tumor-free BW ( C ) in control and KPC tumor–bearing mice. ( D <t>)</t> <t>Four-paw</t> grip strength normalized by tumor-free BW of control and KPC tumor–bearing mice. ( E ) Representative anti-laminin– and DAPI-stained TA muscle transverse sections. Scale bar: 50 μm. ( F ) Quantification of average myofiber cross-sectional area (CSA) in TA muscle of control and KPC tumor–bearing mice. n = 5 per group. All data are presented as mean ± SEM. * P ≤ 0.05, significantly different from control mice injected with PBS alone analyzed by unpaired Student’s t test. ( G ) UMAP plot representing manually annotated clusters for cell type identity. ( H ) Expression of representative marker genes define major cell types in dot plot. ( I ) Split-UMAP illustrating transcriptomic clustering of muscle-derived nuclei of control and KPC tumor–bearing mice. ( J ) Proportion of different nuclei population in GA muscle of control and KPC tumor–bearing mice.
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    ( A ) Schematic representation of experimental design. Twelve-week-old male C57BL/6 were injected with KPC cells or PBS into the pancreas. On day 21, hindlimb muscles of the mice were isolated and processed for morphometric, biochemical, and snRNA-Seq analyses. ( B and C ) Changes in absolute body (BW) weight ( B ), and tumor-free BW ( C ) in control and KPC tumor–bearing mice. ( D <t>)</t> <t>Four-paw</t> grip strength normalized by tumor-free BW of control and KPC tumor–bearing mice. ( E ) Representative anti-laminin– and DAPI-stained TA muscle transverse sections. Scale bar: 50 μm. ( F ) Quantification of average myofiber cross-sectional area (CSA) in TA muscle of control and KPC tumor–bearing mice. n = 5 per group. All data are presented as mean ± SEM. * P ≤ 0.05, significantly different from control mice injected with PBS alone analyzed by unpaired Student’s t test. ( G ) UMAP plot representing manually annotated clusters for cell type identity. ( H ) Expression of representative marker genes define major cell types in dot plot. ( I ) Split-UMAP illustrating transcriptomic clustering of muscle-derived nuclei of control and KPC tumor–bearing mice. ( J ) Proportion of different nuclei population in GA muscle of control and KPC tumor–bearing mice.
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    Image Search Results


    ( A ) Schematic representation of experimental design. Twelve-week-old male C57BL/6 were injected with KPC cells or PBS into the pancreas. On day 21, hindlimb muscles of the mice were isolated and processed for morphometric, biochemical, and snRNA-Seq analyses. ( B and C ) Changes in absolute body (BW) weight ( B ), and tumor-free BW ( C ) in control and KPC tumor–bearing mice. ( D ) Four-paw grip strength normalized by tumor-free BW of control and KPC tumor–bearing mice. ( E ) Representative anti-laminin– and DAPI-stained TA muscle transverse sections. Scale bar: 50 μm. ( F ) Quantification of average myofiber cross-sectional area (CSA) in TA muscle of control and KPC tumor–bearing mice. n = 5 per group. All data are presented as mean ± SEM. * P ≤ 0.05, significantly different from control mice injected with PBS alone analyzed by unpaired Student’s t test. ( G ) UMAP plot representing manually annotated clusters for cell type identity. ( H ) Expression of representative marker genes define major cell types in dot plot. ( I ) Split-UMAP illustrating transcriptomic clustering of muscle-derived nuclei of control and KPC tumor–bearing mice. ( J ) Proportion of different nuclei population in GA muscle of control and KPC tumor–bearing mice.

    Journal: JCI Insight

    Article Title: Fiber-type vulnerability and proteostasis reprogramming in skeletal muscle during pancreatic cancer cachexia

    doi: 10.1172/jci.insight.200396

    Figure Lengend Snippet: ( A ) Schematic representation of experimental design. Twelve-week-old male C57BL/6 were injected with KPC cells or PBS into the pancreas. On day 21, hindlimb muscles of the mice were isolated and processed for morphometric, biochemical, and snRNA-Seq analyses. ( B and C ) Changes in absolute body (BW) weight ( B ), and tumor-free BW ( C ) in control and KPC tumor–bearing mice. ( D ) Four-paw grip strength normalized by tumor-free BW of control and KPC tumor–bearing mice. ( E ) Representative anti-laminin– and DAPI-stained TA muscle transverse sections. Scale bar: 50 μm. ( F ) Quantification of average myofiber cross-sectional area (CSA) in TA muscle of control and KPC tumor–bearing mice. n = 5 per group. All data are presented as mean ± SEM. * P ≤ 0.05, significantly different from control mice injected with PBS alone analyzed by unpaired Student’s t test. ( G ) UMAP plot representing manually annotated clusters for cell type identity. ( H ) Expression of representative marker genes define major cell types in dot plot. ( I ) Split-UMAP illustrating transcriptomic clustering of muscle-derived nuclei of control and KPC tumor–bearing mice. ( J ) Proportion of different nuclei population in GA muscle of control and KPC tumor–bearing mice.

    Article Snippet: Four-paw grip strength of mice was measured using a digital grip strength meter (Columbus Instruments), as previously described ( ).

    Techniques: Injection, Muscles, Isolation, Control, Staining, Expressing, Marker, Derivative Assay